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Astragalus Powdered Extract

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Product Name: Astragalus Powdered Extract
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Astragulus is one of the most prized herbs in Oriental medicine, is actually a member of the pea family. It was first mentioned in the 2,000-year-old classic herbal reference, Shen Nong Ben Cao Jing. It¡¯s Chinese name, huang-qi, means ¡°yellow leader¡± because it is a superior tonic to invigorate vital energy (qi). Astragalus is also a staple of Traditional Chinese Medicine (TCM), and has been shown to reduce the duration and severity of common cold symptoms as well as raise white blood cell counts. Pharmacology:
¡ìAntiviral: It inhibits the replication of coxsackie B-3 virus (CB3V)-RNA, a virus that causes myocarditis in animal models (Peng, 1995).
¡ìAntioxidant: It inhibits lipid peroxidation in rat heart mitochondria (Hong, 1994).
¡ìCardiovascular effects: It increases cardiac output in patients with angina pectoris (Lei, 1994). Astragaluoside IV improves left ventricular end-diastolic volume, left ventricular end-systolic volume and slows heart rate in heart failure. The compoumd also alleviates chest distress and dyspnea associated with heart failure
Hepatoprotective: An ethanol extract of the root of Astraglus alleviated liver injury through a reduction of elevated SGPT levels and subacute toxicity. The herb also decreased loss of righting reflex and protected hepatic cells from pathological changes (Zhang, 1990).
¡ìImmune-stimulating: the herb stimoulates macrophages, promotes antibody formation, and increases T lymphocyte proliferation. F3, an immuno-regulatory component of the herb reverses macrophage suppression induced by urological tumors (Rittenhouse, 1991). Astragalus extracts enhance the antibody response to a T-dependent antigen associated with an increase of Th cell activity in normal and immunodepressed animal models (Zhao, 1990).a fractionated extract of Astragalus potentiates lymphokine-activated killer (LAK) cell ctotoxicity generated by low-dose recombinant interleukin-2 (rIL-2). This immune response occurs through a 10-fold potentiation of rIL-2 acitivity manifested by tumor cell

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